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transwell transparent chamber with an upper chamber coated with matrigel  (Corning Life Sciences)

 
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    Corning Life Sciences transwell transparent chamber with an upper chamber coated with matrigel
    Transwell Transparent Chamber With An Upper Chamber Coated With Matrigel, supplied by Corning Life Sciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/transwell+transparent+chambers/pm40445264-76-41-43?v=Corning+Life+Sciences
    Average 90 stars, based on 1 article reviews
    transwell transparent chamber with an upper chamber coated with matrigel - by Bioz Stars, 2026-08
    90/100 stars

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    transwell chambers with an 8 μm transparent membrane #353182  (Corning Life Sciences)
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    Corning Life Sciences transwell chambers with an 8 μm transparent membrane #353182
    Suppression of Pa03C tumor cell growth by PI3K-activated MSC-derived CM. The single and double asterisks (comparison to CN) and pound signs (comparison to CM control) indicate p < 0.05 and 0.01, respectively. CM conditioned medium, CN control, YS YS49, PI3K activator. ( A , B ) Suppression of <t>transwell</t> invasion of PANC1 and Pa03C cells, respectively, by PI3K-activated MSC-derived CM. ( C ) HE-stained lung sections of NOD/SCID/γ(−/−) mice, showing the suppression of cell growth by YS49-treated MSC CM. Pa03C cells were inoculated from the tail vein (N = 5). ( D ) Fluorescently labeled lung sections of three mouse groups. Pa03C cells were labeled with red fluorescent proteins and all nuclei were stained with DAPI.
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    Suppression of Pa03C tumor cell growth by PI3K-activated MSC-derived CM. The single and double asterisks (comparison to CN) and pound signs (comparison to CM control) indicate p < 0.05 and 0.01, respectively. CM conditioned medium, CN control, YS YS49, PI3K activator. ( A , B ) Suppression of transwell invasion of PANC1 and Pa03C cells, respectively, by PI3K-activated MSC-derived CM. ( C ) HE-stained lung sections of NOD/SCID/γ(−/−) mice, showing the suppression of cell growth by YS49-treated MSC CM. Pa03C cells were inoculated from the tail vein (N = 5). ( D ) Fluorescently labeled lung sections of three mouse groups. Pa03C cells were labeled with red fluorescent proteins and all nuclei were stained with DAPI.

    Journal: Scientific Reports

    Article Title: The inhibition of pancreatic cancer progression by K-Ras-overexpressing mesenchymal stem cell-derived secretomes

    doi: 10.1038/s41598-023-41835-6

    Figure Lengend Snippet: Suppression of Pa03C tumor cell growth by PI3K-activated MSC-derived CM. The single and double asterisks (comparison to CN) and pound signs (comparison to CM control) indicate p < 0.05 and 0.01, respectively. CM conditioned medium, CN control, YS YS49, PI3K activator. ( A , B ) Suppression of transwell invasion of PANC1 and Pa03C cells, respectively, by PI3K-activated MSC-derived CM. ( C ) HE-stained lung sections of NOD/SCID/γ(−/−) mice, showing the suppression of cell growth by YS49-treated MSC CM. Pa03C cells were inoculated from the tail vein (N = 5). ( D ) Fluorescently labeled lung sections of three mouse groups. Pa03C cells were labeled with red fluorescent proteins and all nuclei were stained with DAPI.

    Article Snippet: The invasion ability of cancer cells was determined using 12-well plates (#35-3043, Corning) and transwell chambers with an 8 μm transparent membrane (#353182, Corning).

    Techniques: Derivative Assay, Comparison, Control, Staining, Labeling

    Additive anti-tumor effects of YS49-treated MSC CM and recombinant MSN proteins with Gemcitabine. The single and double asterisks (comparison to CN) and pound signs (comparison to GEM) indicate p < 0.05 and 0.01, respectively. CM conditioned medium, CN control, GEM Gemcitabine. ( A ) MTT-based viability of PANC1 pancreatic cancer cells in response to the condensed CM up to 20-fold. ( B ) Combined effects of 0–5 µM Gemcitabine with 20-fold condensed CM. ( C – E ) Additive anti-tumor effects of 0.5 µM of Gemcitabine and 1 µg/mL of recombinant MSN proteins in the scratch-based motility, EdU-based proliferation, and transwell invasion of PANC1 pancreatic cancer cells, respectively.

    Journal: Scientific Reports

    Article Title: The inhibition of pancreatic cancer progression by K-Ras-overexpressing mesenchymal stem cell-derived secretomes

    doi: 10.1038/s41598-023-41835-6

    Figure Lengend Snippet: Additive anti-tumor effects of YS49-treated MSC CM and recombinant MSN proteins with Gemcitabine. The single and double asterisks (comparison to CN) and pound signs (comparison to GEM) indicate p < 0.05 and 0.01, respectively. CM conditioned medium, CN control, GEM Gemcitabine. ( A ) MTT-based viability of PANC1 pancreatic cancer cells in response to the condensed CM up to 20-fold. ( B ) Combined effects of 0–5 µM Gemcitabine with 20-fold condensed CM. ( C – E ) Additive anti-tumor effects of 0.5 µM of Gemcitabine and 1 µg/mL of recombinant MSN proteins in the scratch-based motility, EdU-based proliferation, and transwell invasion of PANC1 pancreatic cancer cells, respectively.

    Article Snippet: The invasion ability of cancer cells was determined using 12-well plates (#35-3043, Corning) and transwell chambers with an 8 μm transparent membrane (#353182, Corning).

    Techniques: Recombinant, Comparison, Control